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Sabrina Ronen, PhD
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Publications
Sabrina Ronen, PhD's Publications
Imaging the effects of treatment with TERT and EGFR inhibitors on glioblastoma: An MR study.
Hyperpolarized δ-[1- 13C]gluconolactone imaging visualizes response to TERT or GABPB1 targeting therapy for glioblastoma.
Monitoring response to a clinically relevant IDH inhibitor in glioma-Hyperpolarized 13C magnetic resonance spectroscopy approaches.
Telomerase reverse transcriptase induces targetable alterations in glutathione and nucleotide biosynthesis in glioblastomas.
A 13C/31P surface coil to visualize metabolism and energetics in the rodent brain at 3 Tesla.
Deuterium magnetic resonance spectroscopy enables noninvasive metabolic imaging of tumor burden and response to therapy in low-grade gliomas.
Deuterium Metabolic Imaging Reports on TERT Expression and Early Response to Therapy in Cancer.
Imaging biomarkers of TERT or GABPB1 silencing in TERT-positive glioblastoma.
Acquisition and quantification pipeline for in vivo hyperpolarized 13 C MR spectroscopy.
Early Noninvasive Metabolic Biomarkers of Mutant IDH Inhibition in Glioma.
Imaging 6-Phosphogluconolactonase Activity in Brain Tumors In Vivo Using Hyperpolarized δ-[1-13C]gluconolactone.
Metabolic imaging detects elevated glucose flux through the pentose phosphate pathway associated with TERT expression in low-grade gliomas.
Non-invasive assessment of telomere maintenance mechanisms in brain tumors.
Glutamate Is a Noninvasive Metabolic Biomarker of IDH1-Mutant Glioma Response to Temozolomide Treatment.
In vivo detection of γ-glutamyl-transferase up-regulation in glioma using hyperpolarized γ-glutamyl-[1-13C]glycine.
MR-detectable metabolic biomarkers of response to mutant IDH inhibition in low-grade glioma.
Patient-derived cells from recurrent tumors that model the evolution of IDH-mutant glioma.
First hyperpolarized [2-13C]pyruvate MR studies of human brain metabolism.
In vivo investigation of hyperpolarized [1,3-13C2]acetoacetate as a metabolic probe in normal brain and in glioma.
PI3K/mTOR inhibition of IDH1 mutant glioma leads to reduced 2HG production that is associated with increased survival.
2-Hydroxyglutarate-Mediated Autophagy of the Endoplasmic Reticulum Leads to an Unusual Downregulation of Phospholipid Biosynthesis in Mutant IDH1 Gliomas.
HDAC inhibition in glioblastoma monitored by hyperpolarized 13 C MRSI.
Hyperpolarized 13C MRI: Path to Clinical Translation in Oncology.
Late-stage deuteration of 13C-enriched substrates for T1 prolongation in hyperpolarized 13C MRI.
Mutant IDH1 gliomas downregulate phosphocholine and phosphoethanolamine synthesis in a 2-hydroxyglutarate-dependent manner.
Autophagy-Dependent Shuttling of TBC1D5 Controls Plasma Membrane Translocation of GLUT1 and Glucose Uptake.
Clonal expansion and epigenetic reprogramming following deletion or amplification of mutant IDH1.
GLUT3 upregulation promotes metabolic reprogramming associated with antiangiogenic therapy resistance.
Hyperpolarized 13C MR metabolic imaging can detect neuroinflammation in vivo in a multiple sclerosis murine model.
Metabolic Profiling of IDH Mutation and Malignant Progression in Infiltrating Glioma.
Accelerated high-bandwidth MR spectroscopic imaging using compressed sensing.
Detection of inflammatory cell function using (13)C magnetic resonance spectroscopy of hyperpolarized [6-(13)C]-arginine.
Hyperpolarized (13)C MR imaging detects no lactate production in mutant IDH1 gliomas: Implications for diagnosis and response monitoring.
Imaging Renal Urea Handling in Rats at Millimeter Resolution using Hyperpolarized Magnetic Resonance Relaxometry.
Molecular Imaging of Metabolic Reprograming in Mutant IDH Cells.
MR Molecular Imaging of Brain Cancer Metabolism Using Hyperpolarized 13C Magnetic Resonance Spectroscopy.
MR Studies of Glioblastoma Models Treated with Dual PI3K/mTOR Inhibitor and Temozolomide:Metabolic Changes Are Associated with Enhanced Survival.
Mutant IDH1 Expression Drives TERT Promoter Reactivation as Part of the Cellular Transformation Process.
Mutant IDH1 expression is associated with down-regulation of monocarboxylate transporters.
Rapid Conversion of Mutant IDH1 from Driver to Passenger in a Model of Human Gliomagenesis.
1 H-13 C independently tuned radiofrequency surface coil applied for in vivo hyperpolarized MRI.
IDH1 Mutation Induces Reprogramming of Pyruvate Metabolism.
Magnetic Resonance (MR) Metabolic Imaging in Glioma.
Metabolic reprogramming in mutant IDH1 glioma cells.
Metabolic reprogramming of pyruvate dehydrogenase is essential for the proliferation of glioma cells expressing mutant IDH1.
Studies of Metabolism Using (13)C MRS of Hyperpolarized Probes.
Changes in pyruvate metabolism detected by magnetic resonance imaging are linked to DNA damage and serve as a sensor of temozolomide response in glioblastoma cells.
Glioma cells with the IDH1 mutation modulate metabolic fractional flux through pyruvate carboxylase.
HR-MAS MRS of the pancreas reveals reduced lipid and elevated lactate and taurine associated with early pancreatic cancer.
Hyperpolarized [1-13C] glutamate: a metabolic imaging biomarker of IDH1 mutational status in glioma.
MR-detectable metabolic consequences of mitogen-activated protein kinase kinase (MEK) inhibition.
Protein and nucleotide biosynthesis are coupled by a single rate-limiting enzyme, PRPS2, to drive cancer.
Rapid in vivo apparent diffusion coefficient mapping of hyperpolarized (13) C metabolites.
Early Chk1 phosphorylation is driven by temozolomide-induced, DNA double strand break- and mismatch repair-independent DNA damage.
Gene expression profile identifies tyrosine kinase c-Met as a targetable mediator of antiangiogenic therapy resistance.
HDAC inhibition induces increased choline uptake and elevated phosphocholine levels in MCF7 breast cancer cells.
Lactate dehydrogenase A silencing in IDH mutant gliomas.
Non-invasive in vivo assessment of IDH1 mutational status in glioma.
Pyruvate kinase M2 expression, but not pyruvate kinase activity, is up-regulated in a grade-specific manner in human glioma.
Evaluation of heterogeneous metabolic profile in an orthotopic human glioblastoma xenograft model using compressed sensing hyperpolarized 3D 13C magnetic resonance spectroscopic imaging.
Longitudinal evaluation of MPIO-labeled stem cell biodistribution in glioblastoma using high resolution and contrast-enhanced MR imaging at 14.1 tesla.
Treatment with the MEK inhibitor U0126 induces decreased hyperpolarized pyruvate to lactate conversion in breast, but not prostate, cancer cells.
Choline metabolism in malignant transformation.
Detection of early response to temozolomide treatment in brain tumors using hyperpolarized 13C MR metabolic imaging.
Hyperpolarized 13C MR spectroscopic imaging can be used to monitor Everolimus treatment in vivo in an orthotopic rodent model of glioblastoma.
Magnetic resonance spectroscopy detectable metabolomic fingerprint of response to antineoplastic treatment.
Metabolic consequences of treatment with AKT inhibitor perifosine in breast cancer cells.
Reduced phosphocholine and hyperpolarized lactate provide magnetic resonance biomarkers of PI3K/Akt/mTOR inhibition in glioblastoma.
Vascular patterning and permeability in prostate cancer models with differing osteogenic properties.
17-allyamino-17-demethoxygeldanamycin treatment results in a magnetic resonance spectroscopy-detectable elevation in choline-containing metabolites associated with increased expression of choline transporter SLC44A1 and phospholipase A2.
Autophagy facilitates glycolysis during Ras-mediated oncogenic transformation.
Cellular and in vivo activity of a novel PI3K inhibitor, PX-866, against human glioblastoma.
Dynamic nuclear polarization in metabolic imaging of metastasis: common sense, hypersense and compressed sensing.
Hyperpolarized 13C spectroscopic imaging informs on hypoxia-inducible factor-1 and myc activity downstream of platelet-derived growth factor receptor.
Noninvasive detection of target modulation following phosphatidylinositol 3-kinase inhibition using hyperpolarized 13C magnetic resonance spectroscopy.
Fatty acid synthase inhibition results in a magnetic resonance-detectable drop in phosphocholine.
Macromolecular dynamic contrast-enhanced (DCE)-MRI detects reduced vascular permeability in a prostate cancer bone metastasis model following anti-platelet-derived growth factor receptor (PDGFR) therapy, indicating a drop in vascular endothelial growth fa
Monitoring histone deacetylase inhibition in vivo: noninvasive magnetic resonance spectroscopy method.
Detection of histone deacetylase inhibition by noninvasive magnetic resonance spectroscopy.
Identification of magnetic resonance detectable metabolic changes associated with inhibition of phosphoinositide 3-kinase signaling in human breast cancer cells.
Magnetic resonance spectroscopy monitoring of mitogen-activated protein kinase signaling inhibition.
Magnetic resonance spectroscopic pharmacodynamic markers of the heat shock protein 90 inhibitor 17-allylamino,17-demethoxygeldanamycin (17AAG) in human colon cancer models.
Apoptosis is associated with triacylglycerol accumulation in Jurkat T-cells.
Magnetic resonance detects changes in phosphocholine associated with Ras activation and inhibition in NIH 3T3 cells.
Applications of magnetic resonance in model systems: cancer therapeutics.
Imaging biochemistry: applications to breast cancer.
Treatment of SW620 cells with Tomudex and oxaliplatin induces changes in 2-deoxy-D-glucose incorporation associated with modifications in glucose transport.
Magnetic resonance detects metabolic changes associated with chemotherapy-induced apoptosis.
Measurements of human breast cancer using magnetic resonance spectroscopy: a review of clinical measurements and a report of localized 31P measurements of response to treatment.
Comparative NMR study of a differentiated rat hepatoma and its dedifferentiated subclone cultured as spheroids and as implanted tumors.
Lipid metabolism in large T47D human breast cancer spheroids: 31P- and 13C-NMR studies of choline and ethanolamine uptake.
The application of 13C NMR to the characterization of phospholipid metabolism in cells.
Lipid metabolism in T47D human breast cancer cells: 31P and 13C-NMR studies of choline and ethanolamine uptake.
NMR studies of the lipid metabolism of T47D human breast cancer spheroids.
Studies of the metabolism of human breast cancer spheroids by NMR.
